A part of the Office of Undergraduate Research and Scholarships


Information on the Application, Eligibility and Contract Screens for the L&S and Rose Hills Independent and Rose Hills Experience Fellowships are located here

Application with Human Subjects:  2/25/14
w/out Human Subjects: 3/7/14; results announced by 4/15/14

About the Program

The Summer Undergraduate Research Fellowship/L&S program (SURF/L&S) allows UC Berkeley undergraduates in the College of Letters and Science to spend the summer doing concentrated research in preparation for a senior thesis. About forty fellows will be funded in 2014, receiving $4250.  Please fill out the eligibility screen here so we can guide you.

The SURF/Rose Hills fellowship ($6K) allows UC Berkeley students in certain math, science, and engineering majors (L&S, CNR, COE, C. Chem) to immerse themselves in full-time summer research; independent research for juniors or graduating seniors ; or faculty-initiated research for sophomores or juniors. With generous support from The Rose Hills Foundation, 42 students will receive a total of $6,000 each.


The SURF application is now open!  


Additional benefits of both programs

In addition to receiving funding, each summer fellow will be assigned to a research cluster, which will meet weekly throughout the summer. In the cluster you will find a community of undergraduate researchers with similar interests and problems, as well as a cluster mentor, an experienced researcher to help... more

Meet Our SURF & Rose Hills Fellows

Understanding the Effects of AKT Pathway Inhibitors on the Pathogenesis of Mycobacterium Tuberculosis in Macrophages

Robert Potter : Molecular and Cell Biology

Mycobacterium tuberculosis manipulates the host response to prevent bactericidal mechanisms and promote its own survival. AKT is one enzyme that has been linked to proliferation of M. tuberculosis within its target. As a serine/threonine protein kinase, AKT is an important enzyme in signal transduction pathways for apoptosis. Inhibition of AKT during infection leads to increase bacterial death. By looking at the downstream effects of the AKT pathway, our goal is to determine, “why?” This will be accomplished by using site directed mutagenesis to create multiple alterations to the structure of AKT in macrophages and then using inhibitors of AKT, stop the downstream effects at numerous junctures after being infected by a model organism, M. Smegmatis.  Western blotting will be used to... Read More